Abstract:
COVID-19 pneumonia, much like that of bacterial and viral community-acquired pneumonia before it, is accompanied by a high rate
of cardio- and cerebrovascular events that are associated with an increased risk of complications and a greater mortality. Although the
mechanisms underlying the pathogenesis of these adverse events are not entirely clear and may be multifactorial, platelets appear to have a
prominent aetiologic role and this, together with an overview of the clinical evidence, forms the basis of this short review.
