COVID-19 Is a multi-organ aggressor : epigenetic and clinical marks

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Authors

Kgatle, Mankgopo
Lawal, Ismaheel Opeyemi
Mashabela, Gabriel
Boshomane, Tebatso M.G.
Koatale, Palesa Caroline
Mahasha, Phetole Walter
Ndlovu, Honest
Vorster, Mariza
Rodrigues, Hosana Gomes
Zeevaart, Jan Rijn

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Frontiers Research Foundation

Abstract

The progression of coronavirus disease 2019 (COVID-19), resulting from a severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, may be influenced by both genetic and environmental factors. Several viruses hijack the host genome machinery for their own advantage and survival, and similar phenomena might occur upon SARS-CoV-2 infection. Severe cases of COVID-19 may be driven by metabolic and epigenetic driven mechanisms, including DNA methylation and histone/chromatin alterations. These epigenetic phenomena may respond to enhanced viral replication and mediate persistent long-term infection and clinical phenotypes associated with severe COVID-19 cases and fatalities. Understanding the epigenetic events involved, and their clinical significance, may provide novel insights valuable for the therapeutic control and management of the COVID- 19 pandemic. This review highlights different epigenetic marks potentially associated with COVID-19 development, clinical manifestation, and progression.

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Keywords

Cytokine storm, Epigenetics, Multi-organ, Pro-inflammatory cytokines, Coronavirus disease 2019 (COVID-19), COVID-19 pandemic, Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), Angiotensin-converting enzyme 2 (ACE2), Transmembrane serine protease 2 (TMPRSS2)

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Citation

Kgatle, M.M., Lawal, I.O., Mashabela, G., Boshomane, T.M.G., Koatale, P.C., Mahasha, P.W., Ndlovu, H., Vorster, M., Rodrigues, H.G., Zeevaart, J.R., Gordon, S., Moura-Alves, P. & Sathekge, M.M. (2021) COVID-19 Is a Multi-Organ Aggressor: Epigenetic and Clinical Marks. Frontiers in Immunology 12:752380. DOI: 10.3389/fimmu.2021.752380