dc.contributor.advisor |
Anguelov, Roumen |
en |
dc.contributor.coadvisor |
Garba, Salisu M. |
en |
dc.contributor.postgraduate |
Usaini, Salisu |
|
dc.date.accessioned |
2015-11-25T09:53:43Z |
|
dc.date.available |
2015-11-25T09:53:43Z |
|
dc.date.created |
2015/09/01 |
en |
dc.date.issued |
2015 |
en |
dc.description |
Thesis (PhD)--University of Pretoria, 2015. |
en |
dc.description.abstract |
The Allee e ect is characterized by a positive relationship between population density
or size and the per capita population growth rate in small populations. There
are several mechanisms responsible for the Allee e ects. In light of these Allee
mechanisms, we modeled both the birth and the death rates as density-dependent
quadratic polynomials. This approach provides an ample opportunity for taking into
account the major contributors to the Allee e ects and e ectively captures species'
susceptibility variation due to the Allee e ect. We design an SI model with these
demographic functions and show that the host and/or disease persistence and extinction
are characterized by threshold values of the disease related parameters ( and ).
For special cases of the model, veri able conditions for host population persistence
(with or without infected individuals) and host extinction are derived. Numerical
simulations indicate the e ects of the parameter on the host population persistence
and extinction regions.
Furthermore, an SEI model with frequency-dependent incidence and the same
quadratic demographics is presented. This is aimed at investigating the combined
impact of infectious disease and the Allee e ect at higher population levels. Indeed,
the model suggests that the eventual outcome could be an inevitable population crash
to extinction. The tipping point marking the unanticipated population collapse at
high population level is mathematically associated with a saddle-node bifurcation.
The essential mechanism of this scenario is the simultaneous population size depression
and the increase of the extinction threshold owing to disease virulence and the
Allee eff ect. Finally, the role of repeated exposure to mycobacteria on the transmission dynamics
of bovine tuberculosis is addressed. Such exposure is found to induce the
phenomenon of backward bifurcation. Two scenarios when such phenomenon does
not arise are highlighted and for each case the model is proved to have a globally
asymptotically stable equilibrium. The impact of vaccine is assessed via a threshold
analysis approach. |
en |
dc.description.availability |
Unrestricted |
en |
dc.description.degree |
PhD |
en |
dc.description.department |
Mathematics and Applied Mathematics |
en |
dc.description.librarian |
tm2015 |
en |
dc.identifier.citation |
Usaini, S 2015, Epidemiological models with density-dependent demographics, PhD Thesis, University of Pretoria, Pretoria, viewed yymmdd <http://hdl.handle.net/2263/50811> |
en |
dc.identifier.other |
S2015 |
en |
dc.identifier.uri |
http://hdl.handle.net/2263/50811 |
|
dc.language.iso |
en |
en |
dc.publisher |
University of Pretoria |
en_ZA |
dc.rights |
© 2015 University of Pretoria. All rights reserved. The copyright in this work vests in the University of Pretoria. No part of this work may be reproduced or transmitted in any form or by any means, without the prior written permission of the University of Pretoria. |
en |
dc.subject |
UCTD |
en |
dc.title |
Epidemiological models with density-dependent demographics |
en |
dc.type |
Thesis |
en |